Medical tests and procedures can be scary and painful for a child! We can help in using our skills of hypnotherapy and E.F.T. We have the ability to ease their mind and their body. We can help them relax and feel calmer, ease the side effects of treatments including pain and nausea, visualize the cancer being gone and cope in general. Children so easily and effortlessly go into hypnosis. I like to use the idea of a "magic flashlight" with children. They easily go into "the control room" with their active imaginations where the "magic flashlight" can illuminate whatever needs to be found or readjusted or fixed or whatever they come up with.
Cancer can easily cause stress, anxiety or depression. Treatments like chemotherapy, radiation or hormone therapy are all difficult to take and each patient has to adjust to the process. Sometimes concerns so impact daily life that patients develop difficulty adjusting to all that is going on. This means they have problems dealing with everyday living. We are used to using our skills to help our clients regarding these issues and they work so well. It is a known fact that the very act of going into hypnosis will increase your white blood cell count!
Dr. Athar Chishti,wanted to understand how the lipid "PIP3" was sent to where it was needed at the growing tips of the cells. It was determined that a molecular motor protein called a kinesin that they had discovered interacted with PIP3 binding protein. They showed that the kinesin and this binding protein motored the PIP3 along microtubules -- the tracks upon which the kinesins move their cargoes. They also showed that the kinesin-binding adaptor and PIP3 were found together at the tips of the nerve cells It was found that when the adaptor binds PIP3, it is delivered to the membrane where it is needed, and if you block this process, polarity is lost. In some cells, like neurons, there are dramatic differences in the structure and function of the different ends of a cell. Loss of cell polarity is often the first step in converting a normal cell to a cancer cell, Chishti said. This is something I feel we can use to help our cancer patients.
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The latest generation of cancer chemotherapeutic drugs specifically targets mutant enzymes or "oncoproteins" that have run amok and now promote uncontrolled cell growth. Cancer cells, with their backs against the wall, have the tendency to fight back. A major goal of cancer research is to frustrate these acts of cellular desperation. In a forthcoming issue of Cancer Cell, investigators at the Salk Institute for Biological Studies uncover one means cancer cells use to stay alive and in doing so suggest a strategy to overcome their tendency to fight back. The study, shows that resistance to the chemotherapeutic drug rapamycin is mediated by the survival factor NF-kB.
Rapamycin, like the pharmaceutical superstar Gleevec, which revolutionized the treatment of chronic myelogenous leukemia, is a so-called signal transduction inhibitor or STI, a small molecule that stifles inappropriate growth signals sent by mutant proteins in cancer cells. The Hunter lab previously showed that mouse cells lacking tumor suppressors known as TSC genes are more susceptible to the lethal effects of chemotherapeutic agents than are normal cells.
Now, the team found that human cells lacking TSC genes were vulnerable to chemotherapeutic attack because they couldn't activate a major line of defense mediated by the Nuclear Factor kappa B, known as NF-kB, which triggers both inflammatory and survival responses by inducing transcription of specific genes.
Not only did this explain why TSC null cells are vulnerable to insult, but it also provided biochemical evidence that there is crosstalk between two survival mechanisms. The findings show for the first time that the TSC complex can regulate the NF-kB signaling cascade.
The experiments also explained a paradox: TSC null cells treated with rapamycin actually survived cellular insult better than untreated cells-a highly inauspicious outcome if the goal is to kill cancer cells. By increasing NF-kB activity in the TSC null cells when they were exposed to chemotherapeutic drugs.
Rapamycin, an immunosuppressant used to block organ rejection after transplants, also inactivates proteins stimulating cell division and in clinical trials has been combined with other drugs to halt cancer cell growth.
But to cancer cells, rapamycin is both friend and foe. "Rapamycin is not as successful as initially expected in treating cancer," explains Ghosh. "Instead of killing cells, you end up triggering a survival response in them." This study, however, suggests that taking NF-kB out of the game would make rapamycin less "friendly."
A major problem of chemotherapy is that sooner or later cancer cells develop resistance, which requires higher and higher doses of chemotherapeutics. Rapamycin-mediated killing of cancer cells could be increased by inhibiting the function of NF-kB proteins.
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Another piece of research:
Scientists have found a way to trick cancer cells into committing suicide. Most living cells contain a protein called procaspase-3, which, when activated, changes into the executioner enzyme caspase-3 and initiates programmed cell death, called apoptosis. In cancer cells, however, the signaling pathway to procaspase-3 is broken. As a result, cancer cells escape destruction and grow into tumors.
Paul J. Hergenrother, a professor of chemistry at the University of Illinois at Urbana-Champaign and in the publication journal Nature Chemical Biology says, "We have identified a small, synthetic compound that directly activates procaspase-3 and induces apoptosis. By bypassing the broken pathway, we can use the cells' own machinery to destroy themselves."
The researchers tested the compound's efficacy in cell cultures and in three mouse models of cancer. The researchers also showed that PAC-1 killed cancer cells in 23 tumors obtained from a local hospital.
Cell death was correlated with the level of procaspase-3 present in the cells, with more procaspase-3 resulting in cell death at lower concentrations of PAC-1.
So, how does all this apply to our work as hypnotherapists? In our visualizations, we can suggest that the cells are once again finding their top and their bottom. Visualizing the cancer cells just giving up! Talk to the proteins and explain that they can just stop stimulating unwanted cell growth. Let those cancer cells just stop their growth. Visualize activating these executioner enzymes to help the cancer commit suicide. Let's help them to turn the motor on and deliver. Use our clients creativity and our own. The possibilities are endless and as individual as our clients.
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I just have to share some good news that surprised me when I opened my September e-newsletter from Marilyn Gordon's Hypnotherapy Center. Here is the article:
Honoring Our Graduates:
Seth-Deborah Roth
This is the first in our series of honors for the great work of our graduates. Seth-Deborah Roth is our first honoree. She wins this honor for her inspiring, educational work in the field of Medical Hypnotherapy. She teaches in our program and at the National Guild of Hypnotists, and students in her courses have consistently given her rave reviews. Seth's work is exciting, detailed, innovative and creative. She now receives a round of applause and words of appreciation from the Hypnotherapy Center for her inspiring instruction. To find out more about Seth, click
www.hypnotherapyforhealth.com. She also has a marvelous book that you can purchase.
Center for Hypnotherapy
Marilyn Gordon, BCH, CI
1 (800) 398-0034
Thank You Marilyn!
Help spread the word about hypnosis and health! If you know of anyone else who would like to subscribe to the newsletter invite them to click and sign up at: www.hypnotherapyforhealth.com Love and light to all.....
Seth-Deborah
